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Sumption. For example, Simansky et al. w425x found that hypothalamic DOPACrdopamine ratios were increased by conditioned stimuli that ordinarily preceded a meal as much as by the meal itself. Blackburn et al. w52x showed that nucleus accumbens DOPACrdopamine ratios were more highly elevated by conditioned stimuli for food presented alone, without food itself, than by the unexpected opportunity to eat. Using in vivo voltammetry, Phillips et al. found that, during the course of a meal, dopamine release was triggered before the meal by conditioned stimuli, and remained high until after the end of the meal w345347x. Richardson and Gratton similarly reported that, as rats became experienced with the associative relationship between cues and food, dopamine release in the nucleus accumbens shifted forward in time from the presentation of food itself to presentation of conditioned stimuli that had been paired with food w359x. Using voltammetry, Kiyatkin and Gratton w260x found that a dopamine-related signal increased in anticipation of a food reward as trained rats performed a bar press response, and that increments in dopamine activity were time-locked to the goal-directed response in advance of food delivery. Electrophysiological studies by Schultz and colleagues also have shown that dopamine neurons discharge in response to conditioned stimuli predictive of food rewards to a greater extent than when animals actually eat the food Zi.e., before they presumably experience the pleasurable taste of food. In inexperienced monkeys mesolimbic and mesostriatal dopamine neurons discharged only when a palatable liquid was delivered to their mouth or when they were allowed to touch food with their hand w5, 278, 398, 403x. But after a neutral conditioned stimulus Ze.g., light. was repeatedly paired with food, dopamine neurons stopped responding to food itself and instead fired vigorously in response to the newly established conditioned incentie stimulus w5, 278, 398, 403x. In direct contradiction of a `hedonia prediction' the neurons often failed to discharge when an experienced animal actually obtained the sensory pleasure of food in the mouth w450x. Nor was dopaminergic discharge, according to the investigators, coupled to ``mnemonic or preparatory representational task components'' w277x Zp. 337., to the execution of reaching movements to obtain and retrieve food, or to sensory properties of a light unrelated to food w5, 278, 398, 401404, Similarly, Kosobud et al. w269x reported that in rats trained to bar press for sucrose, ventral tegmental area ZVTA. unit activity increased prior to the presentation of sucrose. The discharge of VTA neurons was not correlated with the moment when sucrose actually was in the mouth, when presumably the animal would experience the greatest sensory pleasure produced by the taste of sucrose, but rather preceded it w269x. Based on findings such as those described above, Schultz Z1992, p. 134. concluded ``that dopamine neurons respond specifically to salient stimuli that have alerting, arousing and attention-grabbing properties'' and amoxicillin.

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